Mitochondrial Aβ: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease

ABSTRACT Although amyloid‐β peptide (Aβ) is the neurotoxic species implicated in the pathogenesis of Alzheimer's disease (AD), mechanisms through which intracellular Aβ impairs cellular properties, resulting in neuronal dysfunction, remain to be clarified. Here we demonstrate that intracellular Aβ i... View More